Hemoglobinopathies, Bleeding Disorders & Cancer Genetics

20 questions β€’ 1 test β€’ tap a section to begin

Welcome! 10.5 Hemoglobinopathies, Bleeding Disorders & Cancer Genetics β€” Test 2 — 20 questions, CSIR-NET style.

What this test covers

  • Sickle-cell & thalassaemias (alpha/beta)
  • Bleeding disorders: vWD & haemophilia
  • Cancer genetics: TP53, oncogenes, FAP
  • Achondroplasia, consanguinity & Down screening

How to use

  • Tap the test below — it opens on its own full screen. Use ← All tests at the top to come back.
  • Each question has a 40-second timer. Answer, then Submit to see your score.
  • Tap πŸ“‹ View Solution under any question for a full explanation.

Open Review at the bottom for a quick revision list of every question with its correct answer.

Quiz
Question Palette
Quiz
Question Palette
Quiz
Explanation:

Quick revision: every question with its correct answer. For the full explanation, open the test and tap View Solution.

10.5 Hemoglobinopathies, Bleeding Disorders & Cancer Genetics β€” Test 2
Q1. Sickle-cell disease is caused by a substitution of glutamic acid by valine at position 6 of the:βœ“ Beta-globin chain
Q2. Newborns with sickle-cell disease appear healthy at birth mainly because of the presence of:βœ“ Fetal haemoglobin (HbF)
Q3. Reduced or absent synthesis of beta-globin chains results in:βœ“ Beta-thalassaemia
Q4. A severe form of alpha-thalassaemia causing death in neonates (hydrops fetalis) is:βœ“ Hb Bart hydrops fetalis
Q5. The most common inherited bleeding disorder is:βœ“ Von Willebrand disease
Q6. Which inherited bleeding disorder is autosomal dominant (affecting both sexes)?βœ“ Von Willebrand disease
Q7. Haemophilia A and haemophilia B are inherited as:βœ“ X-linked recessive
Q8. Most hereditary cancer syndromes are inherited in an ___ manner:βœ“ Autosomal dominant
Q9. The most commonly mutated gene in human cancers is:βœ“ TP53 (p53)
Q10. A likely mechanism by which a proto-oncogene is converted into an oncogene is:βœ“ Chromosome translocation (or amplification/point mutation)
Q11. Familial adenomatous polyposis (FAP), which predisposes to colon cancer, is caused by inherited mutation of the:βœ“ APC tumour-suppressor gene
Q12. Fanconi anaemia, which features chromosome breakage and a high cancer risk, results from a defect in:βœ“ A DNA-repair pathway
Q13. A fusion (chimeric) gene created by a chromosome rearrangement is the cause of:βœ“ Chronic myeloid leukaemia (BCR-ABL)
Q14. Achondroplasia, a common form of short-limbed dwarfism, is inherited as:βœ“ Autosomal dominant (FGFR3 mutation)
Q15. Consanguinity (mating between close relatives) most increases the risk of:βœ“ Autosomal recessive disorders
Q16. Increased risk of Down syndrome on maternal serum screening is indicated by:βœ“ Low AFP, low unconjugated estriol and high hCG
Q17. The CF carrier (heterozygote) state is thought to have persisted partly because it may have conferred:βœ“ Resistance to certain diarrhoeal diseases
Q18. In Tay-Sachs and Gaucher disease, the accumulated material builds up because of a defect in:βœ“ Lysosomal enzymes (storage disorders)
Q19. Why do some female carriers of an X-linked recessive disease (e.g. haemophilia) show mild symptoms?βœ“ Skewed (non-random) X-inactivation favouring the mutant X
Q20. Match each condition with its description and select the correct option.βœ“ A-ii, B-i, C-iv, D-iii